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Mechanistically, selenocysteine utilization by gpx4 confers exquisite resistance to irreversible overoxidation as cells expressing a cysteine variant are highly sensitive toward. Beyond the essential role for wt gpx4 to prevent ferroptosis, we also demonstrate that mammalian cells obviously do not require any selenoprotein for proliferation and survival, at. Beyond the essential role for wt gpx4 to prevent ferroptosis, we also demonstrate that mammalian cells obviously do not require any selenoprotein for proliferation and survival, at.
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Mechanistically, selenocysteine utilization by gpx4 confers exquisite resistance to irreversible overoxidation as cells expressing a cysteine variant are highly sensitive toward peroxide. Beyond the essential role for wt gpx4 to prevent ferroptosis, we also demonstrate that mammalian cells obviously do not require any selenoprotein for proliferation. The unique function of gpx4 to reduce complex hydroperoxides including phospholipid and cholesterol hydroperoxides, protects biological membranes from the lipid peroxidation chain.
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